1.α-Synuclein O-GlcNAcylation alters aggregation and toxicity, revealing certain residues as potential inhibitors of Parkinson’s disease

作　　者：Paul M. Levine ;Paul M. Levine ;Ana Galesic 等

出　　处：Proceedings of the National Academy of Sciences. 2019 ;116(5):1511-1519.doi:10.1073/pnas.1808845116

出版社：The National Academy of Sciences of the United States of America

文章 ID：226667350

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2.Polo-like kinase 2 regulates selective autophagic α-synuclein clearance and suppresses its toxicity in vivo

作　　者：Abid Oueslati ;Bernard L. Schneider ;Patrick Aebischer 等

出　　处：Proceedings of the National Academy of Sciences. 2013 ;110(41):E3945-E3954.doi:10.1073/pnas.1309991110

出版社：The National Academy of Sciences of the United States of America

文章 ID：89097059

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3.Synthetic polyubiquitinated α-Synuclein reveals important insights into the roles of the ubiquitin chain in regulating its pathophysiology

作　　者：Mahmood Haj-Yahya ;Bruno Fauvet ;Yifat Herman-Bachinsky 等

出　　处：Proceedings of the National Academy of Sciences. 2013 ;110(44):17726-17731.doi:10.1073/pnas.1315654110

出版社：The National Academy of Sciences of the United States of America

文章 ID：89097290

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4.Induction of de novo α-synuclein fibrillization in a neuronal model for Parkinson’s disease

作　　者：Mohamed-Bilal Fares ;Bohumil Maco ;Abid Oueslati 等

出　　处：Proceedings of the National Academy of Sciences. 2016 ;113(7):E912-E921.doi:10.1073/pnas.1512876113

出版社：The National Academy of Sciences of the United States of America

文章 ID：168413666

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5.Parkin depletion delays motor decline dose-dependently without overtly affecting neuropathology in α-synuclein transgenic mice

作　　者：Margot Fournier ;Amandine Roux ;Jérôme Garrigue 等

出　　处：BMC Neuroscience. 2013 ;14(1):1.doi:10.1186/1471-2202-14-135

出版社：BioMed Central

文章 ID：95150141

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6.Unraveling the complexity of amyloid polymorphism using gold nanoparticles and cryo-EM

作　　者：Urszula Cendrowska ;Paulo Jacob Silva ;Nadine Ait-Bouziad 等

出　　处：Proceedings of the National Academy of Sciences. 2020 ;117(12):6866-6874.doi:10.1073/pnas.1916176117

出版社：The National Academy of Sciences of the United States of America

文章 ID：244844450

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7.The process of Lewy body formation, rather than simply α-synuclein fibrillization, is one of the major drivers of neurodegeneration

作　　者：Anne-Laure Mahul-Mellier ;Johannes Burtscher ;Niran Maharjan 等

出　　处：Proceedings of the National Academy of Sciences. 2020 ;117(9):4971-4982.doi:10.1073/pnas.1913904117

出版社：The National Academy of Sciences of the United States of America

文章 ID：244847043

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8.Polyglutamine expansion affects huntingtin conformation in multiple Huntington’s disease models

作　　者：Manuel Daldin ;Valentina Fodale ;Cristina Cariulo 等

出　　处：Scientific Reports. 2017 ;7(1).doi:10.1038/s41598-017-05336-7

出版社：Springer Nature

文章 ID：235888553

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9.Dissociation of amyloid fibrils of α-synuclein and transthyretin by pressure reveals their reversible nature and the formation of water-excluded cavities

作　　者：Débora Foguel ;Marisa C. Suarez ;Astria D. Ferrão-Gonzales 等

出　　处：Proceedings of the National Academy of Sciences. 2003 ;100(17):9831-9836.doi:10.1073/pnas.1734009100

出版社：The National Academy of Sciences of the United States of America

文章 ID：200588851

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10.Inhibiting transthyretin conformational changes that lead to amyloid fibril formation

作　　者：Scott A. Peterson ;Thomas Klabunde ;Hilal A. Lashuel 等

出　　处：Proceedings of the National Academy of Sciences. 1998 ;95(22):12956-12960.doi:10.1073/pnas.95.22.12956

出版社：The National Academy of Sciences of the United States of America

文章 ID：200603513

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