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  • 标题:Peptidyl–Prolyl cis / trans Isomerase NIMA-Interacting 1 as a Therapeutic Target in Hepatocellular Carcinoma
  • 本地全文:下载
  • 作者:Garam Kim ; Jin Young Kim ; Hong Seok Choi
  • 期刊名称:Biological and Pharmaceutical Bulletin
  • 印刷版ISSN:0918-6158
  • 电子版ISSN:1347-5215
  • 出版年度:2015
  • 卷号:38
  • 期号:7
  • 页码:975-979
  • DOI:10.1248/bpb.b15-00245
  • 出版社:The Pharmaceutical Society of Japan
  • 摘要:

    Phosphorylation of proteins on serine or threonine residues preceding proline is a pivotal signaling mechanism regulating cell proliferation. The recent identification and characterization of the enzyme peptidyl–prolyl cis / trans isomerase never in mitosis A (NIMA)-interacting 1 (PIN1) has led to the discovery of a new mechanism regulating phosphorylation in cell signaling. PIN1 specifically binds phosphorylated serine or threonine residues immediately preceding proline (pSer/Thr-Pro) and then regulates protein functions, including catalytic activity, phosphorylation status, protein interactions, subcellular location, and protein stability, by promoting cis / trans isomerization of the peptide bond. Recent results have indicated that such conformational changes following phosphorylation represent a novel signaling mechanism in the regulation of many cellular functions. Understanding this mechanism also provides new insight into the pathogenesis and treatment of human hepatocellular carcinoma. A better understanding of the role of PIN1 in the pathogenesis of hepatocellular carcinoma may lead to the identification of molecular targets for prevention and therapeutic intervention.

  • 关键词:hepatocellular carcinoma; β-catenin; never in mitosis A (NIMA)-related kinase 6; hepatitis B viral protein; peptidyl–prolyl cis / trans isomerase NIMA-interacting 1
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